12 juillet 2015 Georgette RISSER (INRA Avignon) et la Gariguette : sans ramener sa fraise ... http://www.liberation.fr/sciences/2015/07/05/sans-ramener-sa-fraise_1343859 9 juillet 2015 Interview d'Alain FUCHS, Président du CNRS http://intranet.cnrs.fr/intranet/actus/150707-alain-fuchs-integrite.html 8 juillet 2015 Weigel Lab sur Twitter: "German authorities approve genome edited rape seed (canola) as non-#GMO #goodsenseprevails http://t.co/0XZ6xgfBbG Voir aussi Rapid Trait Development System / RTDS @ CIBUS http://www.cibus.com/about.php 5 juin 2015 Plant architecture and gene network: "Shaping plant architecture" http://dx.doi.org/10.3389/fpls.2015.00233 30 avril 2015 Modélisation de l'hétérosis: A Boolean gene regulatory model of heterosis and speciation. http://www.biomedcentral.com/1471-2148/15/24 28 avril 2015 Cartographie de QTL de résistance du maïs au Grey Leaf Spot (GLS) http://journals.plos.org/plosgenetics/article?id=10.1371/journal.pgen.1005045 "Introduction: Plants resist attack by many plant pathogens by inducing localized cell death. This strategy can provide effective defense against pathogens that require living host tissue (biotrophs or hemibiotrophs) and is the basis for most monogenic resistance. However, it is not effective for pathogens that feed on dead tissue (necrotrophs). For this reason, complete, single-gene resistance is typically unavailable for diseases caused by necrotrophic pathogens. Gray leaf spot (GLS), a foliar disease of maize caused by the polycyclic pathogens Cercospora zeae-maydis and Cercospora zeina, is a necrotrophic pathogen that is mostly controlled by quantitative forms of host plant resistance. Understanding the mechanisms underlying resistance to GLS may further elucidate the biology of host-pathogen interactions since there are contrasting mechanisms of defense against necrotrophic and biotrophic pathogens. GLS is one of the most important maize diseases in the United States and in maize-growing regions worldwide. It can cause yield losses over 70% due to associated severe blighting, stalk deterioration and lodging." Attention: La variabilité génétique pour la résistance prise en compte dans le panel NAM semble assez restreinte: AUDPC (Area Under Disease Progress Curve) entre 36 et 52 22 avril 2015 Résistance du tournesol au mildiou: cartographie de la résistance au mildiou conférée par la lignée HA458 (or. HA 434*3/PI 468435) sur le LG4 http://dx.doi.org/10.1007/s00122-015-2470-8 Carte génétique du LG4 et position de Pl17 http://open-source-biology.com/Breves/LG4_Pl17_mapping.png 22 avril 2015 Population structure and genetic diversity characterization of a sunflower association mapping population using SSR and SNP markers (équipe Argentine) http://dx.doi.org/10.1186/s12870-014-0360-x Liste du matériel génétique analysé http://open-source-biology.com/Breves/s12870-014-0360-x-s6.xlsx 22 avril 2015 Association mapping in sunflower (Helianthus annuus L.) reveals independent control of apical vs. basal branching (USA, University of Georgia, Athens) http://dx.doi.org/10.1186/s12870-015-0458-9 22 avril 2015 Recombination in diverse maize is stable, predictable, and associated with genetic load (USA, Cornell Univ., Buckler Group) http://dx.doi.org/10.1073/pnas.1413864112 22 avril 2015 Une revue sur la biologie de l'interaction {Plasmopara halstedii * Tournesol} et des connaissances acquises à ce jour, en particulier dans le domaine des effecteurs de pathgénicité (France, LIPM - Toulouse) http://dx.doi.org/10.1111/mpp.12164